@article {136,
	title = {Second case of neurocysticercosis in a patient with liver transplantation (first case in Spain): a case report},
	journal = {Transplant Proc},
	volume = {39},
	year = {2007},
	note = {Barra Valencia, V Moreno Elola-Olaso, A Fundora Suarez, Y Meneu Diaz, J C Jimenez de los Galanes, S F Perez Saborido, B San Juan, R Ruiz Gimenez, J Abradelo Usera, M Donat Garrido, M Gimeno Calvo, A Hernandez Perez, M C Jimenez Romero, C Moreno Gonzalez, E Case Reports United States Transplantation proceedings Transplant Proc. 2007 Sep;39(7):2454-7.},
	month = {Sep},
	pages = {2454 - 7},
	edition = {39350},
	abstract = {BACKGROUND: Neurocysticercosis (NCC) is a disorder caused by the Taenia solium larva. It is the most common parasitosis of the central nervous system (CNS). Its distribution is universal, but it is endemic in many developing countries and in the third world. In Spain most patients come from countries where the condition is endemic. However, sporadic cases occur among the population of rural regions. NCC in transplant recipients is uncommon. One renal transplant recipient developed NCC but responded to treatment with praziquantel. Recently, it has been reported to complicate a liver transplantation. CASE REPORT: The patient was a 49-year-old Ecuatorian man who received a cadaveric donor liver graft in June 2001 due to acute liver failure induced by toadstool and was under treatment with FK506. In January 2006, the patient presented with a generalized onset of a tonic-clonic seizure for 1 minute without sphincter incontinence, headache, fever, or previous brain trauma. Neurological evaluation did not show evidence of organic brain dysfunction. The neuroimaging findings (brain) computed tomography scan, magnetic resonance imaging were compatible with NCC: many cystic lesions intra- and extraparenchymatous with a scolex visible in three of them. Serology for cysticercosis in plasma was initially indeterminate but positive afterward. The patient was treated with anticonvulsivants (valproic acid) and albendazole. Systemic steroids were added in order to reduce the edema produced upon death of the cyst. Treatment lasted 3 weeks and it was completed without complications or neurological symptoms. Liver function was not affected. One year later the patient remained asymptomatic. CONCLUSION: NCC is a condition that must be included in the differential diagnosis of patients with CNS involvement and cystic lesions on neuroimaging investigations in transplant recipients, especially patients originating from or traveling to endemic areas. First-line therapy for active cysts includes antiparasitic drugs (albendazole or praziquantel) as well as steroids and anticonvulsivants. In our patient, this therapy was effective.},
	keywords = {*Liver Transplantation, Animals, Brain / radiography, Humans, Liver Failure / parasitology / surgery, Male, Middle Aged, Neurocysticercosis / radiography / *surgery, Spain, Taenia / isolation \& purification, Tomography, X-Ray Computed, Treatment Outcome},
	issn = {0041-1345 (Print) 0041-1345 (Linking)},
	doi = {S0041-1345(07)00883-4 [pii] 10.1016/j.transproceed.2007.07.049},
	author = {Barra Valencia,V. and Moreno Elola-Olaso,A. and Fundora Suarez,Y. and Meneu Diaz,J. C. and Jimenez de los Galanes,S. F. and Perez Saborido,B. and San Juan,R. and Ruiz Gimenez,J. and Abradelo Usera,M. and Donat Garrido,M. and Gimeno Calvo,A. and Hernandez Perez,M. C. and Jimenez Romero,C. and Moreno Gonzalez,E.}
}
